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It is common to be asked to assess problems with a patient’s breathing. Shortness of breath (SOB) has many causes. Most at risk to develop respiratory distress are those who have had thoracic or upper abdominal procedures, and elderly, obese, or poorly nourished patients.
How long has the patient had SOB?
Was the onset slow or rapid?
A rapid onset suggests pneumothorax or pulmonary embolus.
Is the patient cyanotic?
Is the patient already on oxygen (O 2 )? What is the patient’s arterial hemoglobin saturation (Sa O 2 )?
Does the patient have a history of chronic obstructive pulmonary disease (COPD)?
If the patient retains carbon dioxide (CO 2 ), the amount of O 2 that may be delivered is affected.
Has the patient undergone a surgical procedure, and if so, how long ago?
Be especially mindful after placement of central lines.
Are there any other symptoms such as chest pain, cough, or fever?
Are there any changes in the patient’s vital signs?
Measure SaO 2 by pulse oximetry if not already done.
As an alternative, have an arterial blood gas (ABG) kit at the bedside. This will give a rapid indication of the arterial partial pressure of oxygen (PaO 2 ).
Deliver O 2 as necessary.
If the patient does not retain CO 2 , O 2 may be safely delivered at a rate that keeps the O 2 saturation at greater than or equal to 93%. See Table 28.1 for various regimens of O 2 delivery.
System | Recommended Flow | Maximum FiO 2 | Humidification | Control of FiO 2 |
---|---|---|---|---|
Room air | N/A | 0.28 | Poor | N/A |
Nasal cannula | 1–6 L/min | 0.44 | Poor | Poor |
Open face mask | 8–10 L/min | 0.44 | Good | Good |
Venturi mask | 10 L/min | 0.45 | Poor | Great |
Tight face mask | 8–10 L/min | 0.60 | Poor | Good |
Non-rebreather mask | 10 L/min | 1 | Poor | Good |
Endotracheal tube | N/A | 1 | Great | Great |
If the patient has a history of reactive airway disease (RAD; COPD or asthma), order bronchodilator treatment if it has been more than 2 hours since the last treatment ( Table 28.2 ).
Generic | Trade | Dosage | Interval |
---|---|---|---|
β-2 Agonists | |||
Albuterol | Proventil, Ventolin | 2.5–5 mg in 3 mL NS | q4–6h |
2 puffs of MDI | q4–6h | ||
2–4 mg PO | q4–8h | ||
Fenoterol | Berotec | 0.5–1.0 mg in 3 mL NS | q4–6h |
Isoetharine | Bronkosol | 0.3 mg in 2.7 mL NS | q4–6h |
2 puffs of MDI | q4–6h | ||
Metaproterenol | Alupent, Metaprel | 0.3 mg in 2.7 mL NS | q4–6h |
2 puffs of MDI | q4–6h | ||
10 mg PO | q6h | ||
Terbutaline | Brethaire, Bricanyl | 0.25–0.5 mg in 2.6 mL NS | q6–8h |
2 puffs of MDI | q6–8h | ||
2.5–5 mg PO | q6–8h | ||
Anticholinergics | |||
Ipratropium | Atrovent | 0.5 mg in 3 mL NS | q6–8h |
Bromide | 2 puffs of MDI | q6h | |
Atropine | 0.5–1 mg in 3 mL NS | q8h | |
Generic | Trade | Dosage | Interval |
Intravenous Medications | |||
Epinephrine (1:1000) | 0.25–0.5 mg IM | Every 8–12 h | |
Aminophylline (not first-line) | 5 mg/kg IV over | 20–30 min | |
Loading | 0.6–0.9 mg/kg/hrIV (maintain levels at 10–20 mcg/mL) | ||
Maintenance | 6 mg/kg q12h of the long-acting preparation |
Medication | IV Dose | PO Dose | Dose Equivalency a |
---|---|---|---|
Steroids | |||
Hydrocortisone | 250 mg bolus, then 3–5 mg/kg divided q6h | — | 1 |
Methylprednisolone | 1–1.5 mg/kg | — | 5 |
Prednisone | — | 1–1.5 mg/kg | 4 |
Dexamethasone | 0.1–0.2 mg/kg | 5–10 mg | 25–30 |
a Glucocorticoid potency compared with hydrocortisone milligram per milligram.
The patient must be evaluated immediately.
Vital sign trends are important. What has the patient’s HR trend been?
Has the patient’s blood pressure (BP) decreased?
Has the patient required increasing amounts of O 2 ?
What is the patient’s urine output? What is the patient’s fluid balance today and over the duration of the hospitalization?
What are the causes of SOB?
Congestive heart failure (CHF)
Pulmonary embolism (PE)
Cardiac tamponade
Atelectasis (usually 24−48 hours after a surgical procedure)
RAD (e.g., COPD, asthma, or gastroesophageal [GE] reflux)
Upper airway obstruction
Pneumothorax
Pleural effusion
Pneumonia
Aspiration of gastric contents
Anxiety
Marked abdominal distention
Hypoxia
Myocardial infarction (MI), as a cause of SOB or as a sequela of other causes of SOB
PE
Cardiac tamponade
Hypoxia is evident by agitation and cyanosis. Patients with significant hypoxia will appear anxious and ill. CO 2 retention is evident by lethargy or obtundation.
Confirm the patency of the upper airway. Remove any oral obstruction such as dentures or foreign bodies. Tachypnea (respiration rate [RR] >20 breaths/minute) will be present if hypoxia, anxiety, pain, or acidosis is present. Slow RRs (<12 breaths/minute) indicate a depression of the central nervous system (CNS) with medications (such as opiates) or primary cerebral lesion. If there is a cardiac cause, tachycardia or arrhythmia may also be present. Fever denotes an infectious cause but also may be present with atelectasis or PE. A drop in BP may indicate PE, tension pneumothorax, CHF, or sepsis.
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