Temporomandibular Joint Injection


Indications and Clinical Considerations

Injection of the temporomandibular joint is indicated as an important component in the management of temporomandibular joint dysfunction, in the palliation of pain secondary to internal derangement of the joint, and in the treatment of pain secondary to arthritis of the joint. Temporomandibular joint dysfunction (also known as myofascial pain dysfunction of the muscles of mastication ) is characterized by pain in the joint itself that radiates into the mandible, ear, neck, and tonsillar pillars. Headache often accompanies the pain of temporomandibular joint dysfunction and is clinically indistinguishable from tension-type headache. Stress is often the precipitating or exacerbating factor in the development of temporomandibular joint dysfunction. Dental malocclusion may also play a role in the evolution of temporomandibular joint dysfunction. Internal derangement and arthritis of the temporomandibular joint may manifest as clicking or grating when the joint is opened and closed and may be easily heard on auscultation of the opening and closing joint ( Figs. 1.1 and 1.2 ). Other causes of temporomandibular joint pain are listed in Box 1.1 .

FIG. 1.1, Osteoarthritis compared in a specimen (A) radiograph and (B) photograph of a sagittally sectioned specimen. The joint space is narrow and the disk is dislocated anteriorly, with thinning and fraying of the meniscal (m) posterior attachment or bilaminar zone. The condylar head cortex is thickened, with small osteophytes (arrows) . The mandibular fossa is sclerotic and remodeled, and only a shallow concavity is seen where the articular eminence once was.

FIG. 1.2, Internal derangement and arthritis of the temporomandibular joint may manifest as clicking or grating when the joint is opened and closed and may be easily heard on auscultation of the opening and closing joint.

Box 1.1
Common Causes of Temporomandibular Joint Pain and Dysfunction

    • Arthritis

    • Myofascial pain

    • Capsulitis

    • Trauma

    • Myositis

    • Neuralgia

    • Disk derangement

    • Synovitis

    • Tumor

    • Ganglion cyst

    • Malocclusion

Plain radiographs and computed tomography may help identify arthritic changes, with magnetic resonance imaging and ultrasound imaging also being useful in identifying articular disk abnormalities and other abnormalities of the temporomandibular joint ( Figs. 1.3, 1.4, and 1.5 ). If the condition is not promptly treated, the patient may experience increasing pain in the these areas, plus limitation of jaw movement and opening. Recently, the injection of autologous blood and platelet-rich plasma into the temporomandibular joint has gained popularity in the treatment of recurrent temporomandibular joint hypermobility dislocation ( Fig. 1.6 ). This technique is also useful in the injection of other substances into the temporomandibular joint, such as hyaluronic acid derivatives and tenoxicam.

FIG. 1.3, Computed tomography images acquired on a GE high-density 64-slice scanner (GE Healthcare, Cleveland, OH) in spiral acquisition and reformatted in multiplanar reconstructions. A, Left mandible and condyle. B, Right mandible and condyle. The bilateral temporomandibular joints show flattening, lipping, and erosion of the condyle, suggestive of degenerative changes.

FIG. 1.4, Mandibular condyle morphology. A, Normal morphology; B, flattening, defined as loss of the round contour; C, subchondral sclerosis, defined as a condition in which the signal intensity is significantly decreased; D, erosion, defined as an interruption or absence of the cortical lining; and E, osteophyte, defined as marginal hypertrophic bone formation. A–C, Normal morphology and deformities such as flattening and subchondral sclerosis were categorized as normal/mild bony changes. D and E, Erosion and osteophyte were categorized as severe bony changes.

FIG. 1.5, Examples of ultrasound examinations of individuals presenting with A, B, Normal disk position, C, D, reducible disk displacement, and E and F, irreducible disk displacement. A, C, and E, Closed-mouth. B, D, and F, Opened mouth. A–D, Right temporomandibular joint. E and F, Left temporomandibular joint. C , condyle; ias , intra-articular space; sk , skin.

FIG. 1.6, Injection of autologous blood into the temporomandibular joint.

Clinically Relevant Anatomy

The temporomandibular joint is a true joint divided into an upper and a lower synovial cavity by a fibrous articular disk. In health, the disk and muscles allow the joint, muscles, and articular disk to move in concert ( Fig. 1.7 ). The internal derangement of this disk may result in pain and temporomandibular joint dysfunction, but extracapsular causes of temporomandibular joint pain are much more common. The joint space between the mandibular condyle and the glenoid fossa of the zygoma may be injected with small amounts of local anesthetic and corticosteroid. The temporomandibular joint is innervated by branches of the mandibular nerve. The muscles involved in temporomandibular joint dysfunction often include the temporalis, masseter, and external and internal pterygoid, and may include the trapezius and sternocleidomastoid. Trigger points may be identified when these muscles are palpated.

FIG. 1.7, The muscle and joint interaction during (A) opening and (B) closing of the mouth. The relative degree of muscle activation is indicated by the different intensities of red. In B, the superior head of the lateral pterygoid muscle is shown eccentrically active. The locations of the axes of rotation (shown as small green circles in A and B ) are estimates only.

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