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Airway management is the otolaryngologist’s main role in emergencies.
Epiglottitis is an emergency because of the high potential for airway obstruction.
Angioedema involves the reticular dermis as well as the subcutaneous and submucosal layers of nondependent areas.
Malignant otitis externa most commonly affects immunocompromised or elderly patients.
The mylohyoid muscle boundary is crossed in Ludwig’s angina.
In patients with recurrent angioedema, hereditary angioedema must be considered in addition to adverse reaction to medicines such as ACE inhibitors. For hereditary angioedema, C1 esterase inhibitor levels and complement evaluation should be requested (C4).
Ludwig’s angina is an odontogenic infection that arises in the submental and submandibular area and causes swelling of the floor of the mouth with displacement of the tongue posteriorly. Upper airway obstruction can proceed rapidly, making intubation difficult or impossible.
Definitive diagnosis of invasive fungal sinusitis requires histopathologic analysis, which confirms invasion of the fungal elements into the submucosal tissues including vessels, often resulting in necrosis of the involved mucosa and bone. Clinically, the involved tissue often lacks sensation and may appear pale or necrotic.
If a tracheo-innominate bleed occurs in a patient with a cuffed endotracheal or tracheostomy tube, the first step is to overinflate the cuff to tamponade bleeding.
A cricothyroidotomy should be converted to a formal tracheotomy within 24 hours, if possible, to minimize the risk of subglottic stenosis.
A = Airway, B = Breathing, C = Circulation, D = Disability/Drugs (what the patient is taking or what should be given), and E = Exposure/Environmental control.
Etiologies may include but are not limited to: (1) inflammatory changes in the upper airway due to infection, angioedema, or caustic substance exposure, etc.; (2) deep neck space infections; (3) a foreign body within the airway or upper esophagus; (4) blunt or penetrating neck trauma with airway involvement; and (5) bleeding complications including hematoma (posttraumatic or postsurgical).
Epiglottitis is inflammation of the epiglottis, typically due to an infectious etiology resulting in rapid airway obstruction. When the inflammation involves surrounding structures, including the aryepiglottic folds and arytenoid soft tissues, it is referred to as supraglottitis. Mortality rates can reach 20%, making urgent diagnosis and treatment essential. The incidence has rapidly declined since the introduction of Haemophilus influenzae type B vaccination. Commonly a childhood disease in the past, it is now more common in adults. The most common bacteria identified include H. influenzae , beta-hemolytic Streptococcus , Staphylococcus aureus , and Streptococcus pneumoniae . Current belief is that George Washington likely died from acute bacterial epiglottitis.
Children often present with dyspnea, drooling, stridor, or fever. Adults may complain of severe sore throat, odynophagia, and hoarseness. Historically, patients presented acutely but now more patients are presenting in a subacute fashion with slower onset of severe symptoms. The “tripod sign” is classically seen on presentation.
The classic radiographic finding is referred to as the “thumb print sign,” described as swelling of the epiglottis on lateral soft tissue neck x-ray. In children, direct visualization via laryngoscopy in the operating room is recommended. Indirect laryngoscopy (fiber-optic nasopharyngeal laryngoscopy) can be considered in adults if the patient is stable enough to tolerate the procedure. Once the diagnosis is made, treatment should consist of airway management and prompt antibiotic administration. Patients with respiratory distress should be intubated. Patients with respiratory stability may be observed closely (in the ICU) with medical management including antibiotics with activity against H. influenza (second- or third-generation cephalosporin), humidified air, racemic epinephrine, and intravenous steroids. It is important to remember that patients who are being observed should always have equipment for intubation and cricothyroidotomy available at the bedside.
Angioedema is the abrupt onset of nonpitting, nonpruritic edema involving the reticular dermis, subcutaneous, and submucosal layers of nondependent areas. This can affect the lips, soft palate, larynx, and pharynx, causing airway obstruction. Duration typically ranges from 24 to 96 hours. Approximately 25% of the U.S. population will have an episode of urticaria and/or angioedema during their lifetime.
The most common causes of acute angioedema include medications, foods, infections, insect venom, contact allergens (latex), and radiology contrast material. Acute angioedema is arbitrarily defined as symptom duration of less than 6 weeks. The evaluation of chronic angioedema and/or urticaria can be challenging. In the majority of cases, no etiology is ever found.
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