Ulnar Nerve Block at the Elbow


Indications and Clinical Considerations

Cubital tunnel syndrome is caused by compression of the ulnar nerve by an aponeurotic band that runs from the medial epicondyle of the humerus to the medial border of the olecranon ( Fig. 58.1 ). This entrapment neuropathy manifests as pain and associated paresthesias in the lateral forearm that radiate to the wrist and ring and little fingers. If the condition remains untreated, progressive motor deficit and ultimately flexion contracture of the affected fingers can result. The onset of symptoms usually occurs after repetitive elbow motions or from repeated pressure on the elbow, such as that from using the elbows to rise from bed. Direct trauma to the ulnar nerve as it enters the cubital tunnel also may result in a similar clinical presentation.

FIG. 58.1, In cubital tunnel syndrome, the point of maximal tenderness is 1 inch below the medial epicondyle.

Physical findings include tenderness over the ulnar nerve at the elbow. A positive Tinel sign over the ulnar nerve as it passes beneath the aponeuroses is usually present ( Fig. 58.2 ). Weakness of the intrinsic muscles of the forearm and hand that are innervated by the ulnar nerve may be identified with careful manual muscle testing ( Table 58.1 ). Manual muscle testing that assesses the strength of the adductor pollicis includes the Froment and Jeanne signs ( Fig. 58.3 ). Manual muscle testing that tests the interosseous muscles includes the crossed finger test, the finger flexion test, and the Egawa sign ( Fig. 58.4 ). Manual muscle testing that assesses the strength of the lumbar innervated lumbrical muscles includes the Duchenne and André Thomas signs. Manual muscle testing that assesses the strength of the hypothenar muscles includes the Wartenberg, Masse, and Pitres–Testut signs ( Fig. 58.5 ). There is always the possibility that a patient with cubital tunnel syndrome may also have additional ulnar, median, or radial nerve lesions distal to the elbow that may confuse the clinical picture. Furthermore, the clinician should be aware that early in the course of the evolution of cubital tunnel syndrome, the only physical finding other than tenderness over the nerve may be the loss of sensation on the ulnar side of the little finger.

FIG. 58.2, Tinel sign at elbow.

TABLE 58.1
Summary of Ulnar Nerve Motor Signs and Tests Grouped by Affected Musculature
Modified from Goldman SB, Brininger TL, Schrader JW, Koceja DM. A review of clinical tests and signs for the assessment of ulnar neuropathy. J Hand Ther. 2009;22:209–220.
Test Name Description Positive Result
Motor signs involving the adductor pollicis muscle
Froment sign The patient holds a piece of paper using a lateral pinch. The examiner then pulls the paper distally along the thumb’s longitudinal axis and assesses the patient’s method of stabilization. Thumb IP flexion compensates for a weak adductor pollicis muscle.
Jeanne sign The patient holds a piece of paper using a lateral pinch. The examiner then pulls the paper distally along the thumb’s longitudinal axis and assesses the patient’s method of stabilization. Thumb MP hyperextension compensates for a weak adductor pollicis muscle.
Motor signs and tests involving the interosseous muscles
Finger flexion sign Performed bilaterally at the same time. Both forearms and wrists are in neutral. Examiner first places a piece of paper between the middle and ring fingers in both hands and then pulls the paper distally. The involved side will use MP flexion to compensate for interossei weakness.
Crossed finger test Examiner asks the patient to cross the middle finger over the index finger. Inability to cross the fingers. Compare with uninvolved side.
Egawa sign Examiner asks the patient to flex the middle finger MP joint and then abduct it to both sides. This can be difficult to perform; therefore, bilateral assessment is recommended. Inability to perform this action as compared with uninvolved side.
Motor signs involving the ulnar nerve—innervated lumbrical muscles
Duchenne sign Sign is identified by observing the posture of the small and ring fingers on the involved side. Clawing posture (MP hyperextension and IP flexion) present in the ring and small fingers.
André Thomas sign Sign is identified by observing the compensatory pattern used in the ring and small fingers during actions involving EDC use. Wrist tends to flex with ring and small finger EDC activation.
Motor signs involving the hypothenar musculature
Wartenberg sign Patient actively abducts the fingers with the forearm in pronation and the wrist in neutral. Observe the small finger’s ability to fully adduct. Inability of the small finger to fully adduct and touch the ring finger. Compare with the uninvolved side.
Masse sign Observe the metacarpal arch as compared with the uninvolved side. The convex nature of the ulnar aspect of the hand is altered owing to hypothenar atrophy. Flattened metacarpal arch.
Pitres–Testut sign Noted after the examiner asks the patient to shape the hand in the form of a cone. Although present in the literature, this sign is not commonly used in clinical practice settings. Inability to shape the hand in the form of a cone.
Palmaris brevis sign A rarely observed sign in lower ulnar nerve palsy in which the lesion selectively affects the deep branch. Determine the presence of this sign by observing and evaluating the palmaris brevis muscle compared with the uninvolved side. The sparing of the palmaris brevis muscle compared with the uninvolved side.
Motor signs involving the extrinsic ulnar nerve–innervated muscles
Nail file sign Patient attempts to make a hook fist. Examiner places an index finger along the volar surface of the patient’s small and ring fingers, leaving the DIPs free to contract. Decreased small and ring finger FDP strength as compared with the uninvolved side.
DIP, Distal interphalangeal; EDC, extensor digitorum communis; FDP, flexor digitorum profundus; IP, interphalangeal; MP, metacarpophalangeal.

FIG. 58.3, Positive Froment sign on the left, negative Froment sign on the right. It is recommended that the wrist be positioned in slight flexion when this test is performed.

FIG. 58.4, Crossed finger test. The left hand shows a positive crossed finger test result because the index finger is unable to completely cross over the middle finger. The right hand shows a negative crossed finger test result.

FIG. 58.5, Wartenberg sign. The right hand indicates a positive Wartenberg sign because the small finger is unable to fully adduct and touch the ring finger. The left hand indicates a negative Wartenberg sign.

Cubital tunnel syndrome often is misdiagnosed as “golfer’s elbow,” and this accounts for the many patients whose golfer’s elbow fails to respond to conservative measures. Cubital tunnel syndrome can be distinguished from golfer’s elbow in that in cubital tunnel syndrome the maximal tenderness to palpation is over the ulnar nerve 1 inch below the medial epicondyle, whereas in golfer’s elbow the maximal tenderness to palpation is directly over the medial epicondyle (see Fig. 58.1 ). Cubital tunnel syndrome also should be differentiated from cervical radiculopathy involving the C7 or C8 roots and golfer’s elbow. Furthermore, it should be remembered that cervical radiculopathy and ulnar nerve entrapment may coexist as the so-called “double crush” syndrome, which is seen most often with median nerve entrapment at the wrist or with carpal tunnel syndrome.

Electromyography helps distinguish cervical radiculopathy and cubital tunnel syndrome from golfer’s elbow. Plain radiographs, ultrasound imaging, and magnetic resonance imaging (MRI) are indicated for all patients with cubital tunnel syndrome to rule out occult bony disease and to confirm the clinical diagnosis ( Fig. 58.6 ). On the basis of the patient’s clinical presentation, additional testing may be indicated, including complete blood count, uric acid, sedimentation rate, and antinuclear antibody testing. MRI and/or ultrasound imaging of the elbow is indicated if joint instability is suspected. The injection technique described later serves as both a diagnostic and a therapeutic maneuver.

FIG. 58.6, Ulnar entrapment neuropathy. Ulnar neuropathy secondary to degenerative arthritis of the elbow. A and B, Axial T2 fat-suppressed image of the elbow show edema of the ulnar nerve in the cubital tunnel (arrows) from compression by large osteophytes arising from the anteromedial surface of the olecranon ( arrowhead , A ) and the medial surface of the olecranon fossa of the humerus ( arrowhead , B ).

Clinically Relevant Anatomy

The ulnar nerve is made up of fibers from the C6-T1 spinal roots. The nerve lies anterior and inferior to the axillary artery in the 3 o’clock to 6 o’clock quadrant. Exiting the axilla, the ulnar nerve descends into the upper arm along with the brachial artery. At the middle of the upper arm, the nerve courses medially to pass between the olecranon process and medial epicondyle of the humerus. It is at this point that the entrapment of the ulnar nerve responsible for cubital tunnel syndrome occurs. The nerve then enters the cubital tunnel and passes between the heads of the flexor carpi ulnaris muscle, continuing downward, moving radially along with the ulnar artery. At a point approximately 1 inch proximal to the crease of the wrist, the ulnar nerve divides into the dorsal and palmar branches. The dorsal branch provides sensation to the ulnar aspect of the dorsum of the hand, the dorsal aspect of the little finger, and the ulnar half of the ring finger. The palmar branch provides sensory innervation to the ulnar aspect of the palm of the hand, the palmar aspect of the little finger, and the ulnar half of the ring finger.

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