Arthroscopic remplissage for management of engaging and deep hill-sachs lesions


OVERVIEW

Chapter synopsis

  • Arthroscopic Hill-Sachs remplissage (AHSR) is indicated for patients who are at high risk for recurrence or failure after isolated arthroscopic anterior soft tissue repair, and in whom large humeral head defects are contributing significantly to the instability complex.

Important points

  • Indications: Anterior shoulder instability with associated large Hill-Sachs defects as visualized on preoperative plain radiographs, computed tomography (CT) scanning, or magnetic resonance imaging (MRI), or identified as engaging the glenoid surface during diagnostic arthroscopy at the time of instability surgery.

  • Contraindications: Isolated glenoid bone loss without a Hill-Sachs defect, unaddressed glenoid bone loss greater than 10%, or forms of shoulder instability other than isolated traumatic anterior instability.

  • Surgical technique: AHSR is not indicated as an isolated procedure and is typically performed in combination with an anterior capsulolabral repair or bone-block augmentation of the anterior glenoid as indicated.

Clinical and surgical pearls

  • Adequate preoperative workup is important to fully assess the size and location of osseous deficiency on both the humeral and glenoid sides of the shoulder joint. Hill-Sachs lesions can be classified as “on-track” versus “off-track” to aid in the understanding of whether the humeral defect will engage on the glenoid rim during shoulder motion.

  • An accessory posterolateral portal is established through which the base of the lesion is debrided and suture anchors are inserted.

  • The anterior repair must be prepared before proceeding to the posterior procedure, as the working space within the joint is significantly reduced after remplissage.

  • Once the humeral sutures have been placed, it is not necessary to reposition the arthroscope in the posterior subdeltoid space before knot tying. More important is the ability to visualize the quality of reduction of the posterior capsulotenodesis into the defect from within the joint as the sutures are being tied.

Clinical and surgical pitfalls

  • After preparation of the anterior capsulolabral tissues, the humeral head is translated anteriorly and perched on the glenoid rim. The Hill-Sachs defect is prepared and anchors are inserted into its base via an 8-mm cannula placed through the posterolateral accessory portal. Extreme care should be taken to ensure an optimal trajectory of suture anchor insertion. If a poor line is taken, there is a risk of anchor penetration of the anterior humeral head cartilaginous surface.

  • Neither of the two suture limbs exiting via the posterolateral portal should be used as the posts during arthroscopic knot tying, as the resultant knot will end up within the joint and the quality of remplissage will be reduced.

Video available

  • Arthroscopic Bankart repair and Hill-Sachs remplissage

Recurrent anterior instability rates of up to 15% have been reported after arthroscopic labral repair for anterior shoulder instability. Patient age and activity level, soft tissue quality, and the presence of glenoid or humeral bone loss have been identified as predisposing factors. A number of potential therapeutic options exist for the management of humeral lesions that are contributing significantly to the instability complex, including open matched osteoarticular allograft transplantation, transhumeral bone grafting, humeroplasty, rotational humeral osteotomy, and partial or complete humeral head resurfacing.

In arthroscopic Hill-Sachs remplissage (AHSR), a procedure pioneered by Wolf and adapted from the open technique described by Connolly, the infraspinatus tendon and posterior capsule are used to fill the humeral defect. The term remplissage derives from the French verb remplir, meaning “to fill.” This posterior capsulotenodesis is not intended to be an isolated procedure, but rather is always performed in combination with an anterior soft tissue repair or bone-block augmentation as indicated according to the “on-track/off-track” concept. Through “filling” of the defect, the defect is rendered extra-articular and is prevented from engagement with the glenoid. In addition, the infraspinatus tendon and capsule act as a checkrein, preventing anterior humeral head translation ( Fig. 14.1 ).

Fig. 14.1
Biomechanics of Hill-Sachs Lesion and Hill-Sachs Remplissage.
(A) Impaction of the posterior humeral head on the anterior glenoid rim (arrow) produces the engaging Hill-Sachs lesion. (B) Recurrence of anterior instability despite anterior Bankart repair because of engagement of the large Hill-Sachs lesion on the anterior glenoid rim (arrow) . (C) The transfer (and healing) of the capsule and infraspinatus tendon into the bone defect has the dual effects of (1) placing the humeral bony lesion extra-articularly (exclusion effect), and (2) decreasing anterior translation (checkrein effect).

Preoperative considerations

History and physical examination

A complete history of the mechanism and frequency of shoulder dislocations should be discussed with the patient. Because the Hill-Sachs lesion represents a humeral head impaction fracture, the history is typically that of joint dislocation rather than repeated episodes of subluxation. The specific sporting activity and level to which it is played should be recorded.

Physical examination is per the standard for anterior shoulder instability. The location of the humeral defect will predict the position of apprehension and engagement. Defects with a more vertical orientation tend to engage with the arm positioned by the side.

Signs of anterior and inferior apprehension and hyperlaxity are sought. Anterior hyperlaxity is present if the examiner can easily subluxate the humeral head out of the socket in the anteroposterior (AP) direction on drawer testing or if passive external rotation is greater than 85 degrees with the patient’s arm at his or her side. Inferior hyperlaxity is assessed with sulcus sign testing and the hyperabduction test, the result of which is positive with a minimum of 20 degrees of asymmetrical abduction at the glenohumeral joint. The presence of skin striae can be a subtle but useful finding suggestive of a predisposition toward soft tissue laxity.

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