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Despite attempts to reduce the incidence of ankle sprain injuries worldwide, ankle sprains continue to account for a large proportion of sport-related injuries as reported consistently in epidemiological studies. In the US population in general, the incidence of ankle sprains was reported to be 2.15 per 1000 person-years with the peak incidence in ages 15–19 (7.2 per 1000 person-years). Almost 50% of those ankle sprains happened in athletic endeavors, with basketball, football, and soccer having the highest percentage of ankle sprain injuries among sports in the United States. Hootman et al. reported that ankle sprains accounted for 15% of all injuries in 15 sports analyzed using National Collegiate Athletic Association (NCAA) injury surveillance data from 1988–2004. They found ankle sprains to have an incidence of 0.83 per 1000 athlete exposures (AEs) compared with 0.15 per 1000 AEs for anterior cruciate ligament (ACL) injury and 0.28 per 1000 AEs for concussions. Thus, an ankle sprain is approximately five and a half and three times as common in occurrence as an ACL or concussion, respectively!
In an epidemiological study of ankle injuries in 255 countries, Fong et al. reported on 70 sports, with aeroball, wall climbing, and indoor volleyball having the highest prevalence of ankle injury, with ankle sprains accounting for over 50% of the ankle injuries in 30 of the 43 studies where injury type was defined. A systematic review and meta-analysis of prospective epidemiological studies was reported by Doherty et al. and concluded that higher risk for ankle sprain occurred in (1) females over males, (2) children over adolescents and adults, (3) court sports and indoor sports, and (4) lateral sprains over syndesmosis and medial sprains.
Ankle sprains include those to the lateral ligament complex, the deltoid complex, and the syndesmosis. Sprains of the lateral ankle ligament complex generally comprise up to 85% of all sprains to the ankle. It is important to realize that while this chapter is separated for discussion of these specific areas of injury, combination injuries occur and include multiple sprains, as well as other injuries including fractures, contusions, strains, tendon tears, cartilage injury, and tendon dislocations. Clearly, this requires a thorough evaluation of each patient to understand the mechanism of injury, the severity and nature of the symptoms, and any history of ankle problems. Coupling this with a detailed physical examination and appropriate imaging allows determination of the best treatment for the individual patient. Postinjury follow-up is also important, since persistent symptoms can exist in 32%‒74% of individuals with a history of lateral ankle sprain, and these symptoms may continue beyond 10 years. A checklist of potential sources of continuing symptoms is valuable ( Table 15.1 ).
Articular injury | Impingement |
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Nerve injury | Miscellaneous conditions |
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Other ligamentous injury | Unsuspected rheumatologic condition |
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Approximately 70% of all lateral ankle sprains involve isolated anterior talofibular ligament (ATFL) injury. The ATFL is the weakest of the lateral ligament structures and is put under increased stress in the most common mechanism of injury, which is foot supination with heel inversion while the ankle is plantarflexed. Pain and swelling are present followed by bruising, loss of motion, and difficulty walking in more serious cases. The anterior drawer and talar tilt tests signify instability when positive, and this may vary from mild to severe. A classification of lateral ankle sprains related to the severity of injury and magnitude of the above findings has been published and validated for time to recovery ( Table 15.2 ).
Grade | Clinical Tests | Decreased ROM | Edema | Stress Radiographs | Return to Sport (days) | ||
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I | <5° | <0.5 cm | Normal | 7.24 ±1.63 | |||
II | + anterior drawer | 5–10° | 0.5–2 cm | Normal | 14.95 ± 2.1 | ||
IIIA | + anterior drawer & talar tilt | >10° | >2 cm | Normal | 30.65 ± 3.07 | ||
IIIB | + anterior drawer & talar tilt | >10° | >2 cm | Joint laxity >3 mm | 55.41 ± 4.92 |
Acute lateral ankle sprains are typically treated nonoperatively (see Chapter 14 ) in almost all circumstances. Exceptions to this would include open injuries, large avulsion fractures, or other associated pathology such as dislocating peroneal tendons, an osteochondral fracture or loose body, or a bimalleolar fracture equivalent, which would essentially be a dislocated ankle that tears both medial and lateral ligaments. Among the remaining controversies related to acute lateral ankle sprains is whether to operate when faced with an athlete who clearly has a severe injury with major instability. Varying opinions can still be heard on this topic. Another situation that stimulates discussion over whether to operate is the athlete who has a history of significant prior ankle sprains and then presents with a recurrent severe sprain. This is the situation that requires a thorough discussion with the patient to determine the best course of action, and extenuating circumstances often play a role. In other words, is this the correct time to repair what is clearly chronic lateral ankle instability?
When nonoperative treatment is the chosen course, a comprehensive plan needs to be drafted that includes evidence-based interventions. This includes functional rehabilitation with a supportive ankle brace in Grade I and II injuries, while Grade III injuries are more controversial regarding functional treatment versus a short period of immobilization in a walking boot or short-leg cast for 10–14 days. Additional treatment includes rest, ice, compression, and elevation method (RICE), antiinflammatory medication (if there are no contraindications), physical therapy with supervised early exercise when possible, and crutches, but only when weight bearing is not possible. In this latter situation, it is important to immobilize the ankle in neutral to slight dorsiflexion, or the ankle will naturally assume a plantarflexed position and heal with the ligaments in an elongated state. Improvement in ankle dorsiflexion, increased strength in the kinetic chain, manual therapy techniques, and balance training have all been shown to have evidence-based gains in ankle function and pain. Other treatments with less evidence include laser therapy, dry needling, vascular restriction, electrotherapy, vascular restriction, diathermy, and ultrasound. The majority of patients treated with evidence-supported methods improve relatively quickly. According to the classification of acute lateral ankle sprains as Grade I, II, IIIA, or IIIB, full recovery averages 8, 16, 25, and 39 days, respectively. However, when this is not the case, further evaluation is warranted, particularly in the high-level athlete where time loss is critical.
In the situation where surgical repair of the torn ligaments is necessary, it is important to define the tissues and repair them anatomically whenever possible ( Figs. 15.1 and 15.2 ). In the acute situation, the injured tissues typically become immediately evident on opening the skin incision. It is important to assess all local structures including the articular cartilage of the ankle joint, the anterior capsule, the deltoid ligament, the peroneal tendons, and superior peroneal retinaculum. At the same time, the surgeon should be protective of the superficial nerves.
In severe sprains, which are essentially dislocations of the ankle joint, one or more of these structures may be injured and need repair. Ligaments torn in midsubstance are repaired with sutures, while tears off bone or with a bony avulsion are repaired with suture anchors. Newer methods of augmentation of repairs have been introduced and proven to be effective. In cases of acute on chronic tears, tissue quality may be lacking and repair with augmentation may be more critical to obtain adequate stability versus reconstruction with a tendon autograft or allograft or a tendon transfer (see next section). The rehabilitation program in these cases is very important and is outlined in the section on rehabilitation ( Boxes 15.1 to 15.3 and Tables 15.3 to 15.5 ).
Use objective outcome measures throughout recovery. Recommend FAAM, FAOS, SANE, VAS scores.
Little to no edema, recommend Figure of Eight method, ICC = 0.99 (Tatras–Adams)
Symmetrical AROM and PROM
Weight Bearing Lunge Test, achieve at least 50% as compared with uninvolved side. Measure distance of foot from wall in cm and tibial angle with inclinometer. ICC = 0.96 to 0.99 (Konor et al.).
Demonstrate proper open chain muscle recruitment for all lower extremity musculature through full available range of motion. Patient can resist increasingly difficult resistance bands × 15 reps through full ROM without pain.
Demonstrate ability to perform short arch exercise in seated position, and intrinsic flexion exercises (McKeon et al.)
Patient can ambulate with safe and appropriate gait pattern in boot without crutches.
Objective outcome measures score reassessed.
Normal Gait pattern demonstrated at varied cadence on flat surface.
Foot Lift Test, demonstrate <5 errors during testing period (Linens et al.)
Weight-Bearing Lunge Test, within 75% of uninvolved leg.
Standing Double-Leg Heel Raise, can demonstrate equal heel height.
Double-Leg Squat, demonstrate proper technique.
Isotonic Single-Leg Leg Press: Achieve 50–60% of body weight × 15 reps
Demonstrate ability to perform seated towel curls with added weight, pulling weight equal to capability of uninvolved side
Criteria for progression met. If any goals are not achieved, this is reviewed and discussed with rehabilitation team and physician.
Subjective outcome measure reassessed; progress suggests return-to-sport readiness. SANE score >93%.
Athlete reports he/she has resumed preinjury training volume.
Return to practice noncontact × 1 week
Return to practice contact × 1 week
Return to game play, progressively working up to preinjury position time of play.
Procedure(s) | Brostrӧm Repair | Brostrӧm Repair + InternalBrace | ATFL and CFL Reconstruction With Allograft | Syndesmosis Repair of AITFL and Tight Rope | Syndesmosis Repair, Deltoid Repair and Fibular ORIF (no cartilage injury) |
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Range-of-Motion Precautions | No inversion or plantarflexion × 6 weeks |
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None | No eversion × 6 weeks |
Weight-Bearing Orders |
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Return to Sport |
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Clinical Finding or Test |
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Test | Passing Score | Notes |
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Weight Bearing Lunge Test | Symmetry to uninvolved side, within 5∗ or 1.5 cm | ICC = 0.96 to 0.99 (Konor et al.) |
Heel Raise Endurance Test | Within 90% of uninvolved side, recorded in successful repetitions | ICC = 0.97 (Sman et al.) |
Single Leg Squat Endurance Test | Within 90% of uninvolved side, recorded in repetitions | ICC = 0.95–1.0 (Garrison et al.) |
Y Balance Test | Composite score above 90% Anterior reach within 4 cm of uninvolved side |
ICC= 0.85–0.93 (Shaffer et al.) |
Isotonic Leg Press | Within 90% of uninvolved 3 × 5 reps |
Clinical Finding or Test |
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Test | Passing Score | Notes |
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Single hop for distance Triple hop for distance |
Within 90% of uninvolved side | ICC = 0.92–0.97 (Ross et al.) ICC = 0.84–0.98 (Kockum et al.) |
Square Hop Figure of 8 Hop Side Hop Cross-Over Hop |
Within 90% of uninvolved side, measured in seconds | Square Hop: ICC = 0.90 SEM 1.40 sec MDC 3.88 sec Side Hop: ICC = 0.84 SEM 2.10 sec MDC 5.82 sec 6 Meter Cross Over Hop: ICC = 0.6 SEM 0.37 sec MDC 1.03 sec (Caffrey et al.) |
MAT Test Modified T Test |
Within 90% of uninvolved side, measured in seconds | ICC = 0.825 (Hickey et al.) |
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