Injection Technique for Gluteus Medius Syndrome


Indications and Clinical Considerations

The gluteus medius muscle is susceptible to developing myofascial pain syndrome. Such pain most often occurs as a result of repetitive microtrauma to the muscle from activities such as running on soft surfaces and overuse of exercise equipment or other repetitive activities that require hip abduction. Blunt trauma to the muscle may also incite gluteus medius myofascial pain syndrome.

Myofascial pain syndrome is a chronic pain syndrome that affects a focal or regional portion of the body. The sine qua non of myofascial pain syndrome is the finding of myofascial trigger points on physical examination. Although these trigger points generally are localized to the regional part of the body affected, the pain of myofascial pain syndrome often is referred to other anatomic areas. This referred pain often is misdiagnosed or attributed to other organ systems, leading to extensive evaluations and ineffective treatment. Patients with myofascial pain syndrome involving the gluteus medius often have primary pain along the posterior iliac crest referred down the buttocks across the sacroiliac joint and into the posterior lower extremity ( Fig. 134.1 ).

FIG. 134.1, Trigger point location and pattern of referred pain in patients with gluteus medius syndrome. m., Muscle.

The trigger point is the pathognomonic lesion of myofascial pain and is thought to be a result of microtrauma to the affected muscles. This pathologic lesion is characterized by a local point of exquisite tenderness in affected muscle. Mechanical stimulation of the trigger point by palpation or stretching produces not only intense local pain but also referred pain. In addition to this local and referred pain, there often is an involuntary withdrawal of the stimulated muscle, called a “jump sign.” This jump sign also is characteristic of myofascial pain syndrome. Patients with gluteus medius syndrome will exhibit a trigger point along the posterior iliac crest (see Fig. 134.1 ).

Taut bands of muscle fibers often are identified when myofascial trigger points are palpated. In spite of this consistent physical finding in patients with myofascial pain syndrome, the pathophysiology of the myofascial trigger point remains elusive, although many theories have been advanced. Common to all of these theories is the belief that trigger points are a result of microtrauma to the affected muscle. This microtrauma may occur as a single injury to the affected muscle or as a result of repetitive microtrauma or chronic deconditioning of the agonist and antagonist muscle unit.

In addition to muscle trauma, a variety of other factors seem to predispose the patient to develop myofascial pain syndrome. The weekend athlete who subjects his or her body to unaccustomed physical activity may develop myofascial pain syndrome. The poor posture of someone sitting at a computer keyboard or while watching television has also been implicated as a predisposing factor to the development of myofascial pain syndrome. Previous injuries may result in abnormal muscle function and predispose the patient to the subsequent development of myofascial pain syndrome. All of these predisposing factors may be intensified if the patient also has poor nutritional status or coexisting psychological or behavioral abnormalities, including chronic stress and depression. The gluteus medius muscle seems to be particularly susceptible to stress-induced myofascial pain syndrome.

Stiffness and fatigue often coexist with the pain of myofascial pain syndrome, increasing the functional disability associated with this disease and complicating its treatment. Myofascial pain syndrome may occur as a primary disease state or in conjunction with other painful conditions, including radiculopathy and chronic regional pain syndromes. Psychological or behavioral abnormalities, including depression, frequently coexist with the muscle abnormalities associated with myofascial pain syndrome. Treatment of these psychological and behavioral abnormalities must be an integral part of any successful treatment plan for myofascial pain syndrome.

Clinically Relevant Anatomy

The primary function of the gluteus medius muscle is as a hip abductor, and it also assists in medial and lateral rotation of the hip. The muscle’s origin is at the dorsal ilium just below the iliac crest (see Fig. 134.1 ), and it inserts on the greater tuberosity of the femur. The muscle is innervated by the superior gluteal nerve ( Fig. 134.2 ). The gluteus medius muscle is susceptible to trauma and to wear and tear from overuse and misuse and may develop myofascial pain syndrome, which may also be associated with gluteal bursitis and acute muscle strain ( Fig. 134.3 ).

FIG. 134.2, Possible entrapment of the superior gluteal nerve. A, Transverse, T1-weighted, spin-echo magnetic resonance imaging (MRI) shows denervation hypertrophy of the tensor fasciae latae muscle (arrow). B, Similar hypertrophy and high signal intensity are seen in the muscle (arrow) on transverse, fat-suppressed, T1-weighted, spin-echo MRI obtained after intravenous gadolinium administration.

FIG. 134.3, A, Magnetic resonance imaging, axial slice: large area of T2 hypersignal indicates inflammation around the detached and retracted lateral fibers of the gluteus medius (GMe) and the subgluteus minimus bursa. B, Coronal slice: GMe tendon tear and bursitis. C, Sagittal slice: the main (posterior) tendon of the GMe is intact.

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